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contributor authorB. A. Younis
contributor authorS. A. Berger
date accessioned2017-05-09T00:12:16Z
date available2017-05-09T00:12:16Z
date copyrightOctober, 2004
date issued2004
identifier issn0148-0731
identifier otherJBENDY-26391#578_1.pdf
identifier urihttp://yetl.yabesh.ir/yetl/handle/yetl/129569
description abstractDifficulties in predicting the behavior of some high Reynolds number flows in the circulatory system stem in part from the severe requirements placed on the turbulence model chosen to close the time-averaged equations of fluid motion. In particular, the successful turbulence model is required to (a) correctly capture the “nonequilibrium” effects wrought by the interactions of the organized mean-flow unsteadiness with the random turbulence, (b) correctly reproduce the effects of the laminar-turbulent transitional behavior that occurs at various phases of the cardiac cycle, and (c) yield good predictions of the near-wall flow behavior in conditions where the universal logarithmic law of the wall is known to be not valid. These requirements are not immediately met by standard models of turbulence that have been developed largely with reference to data from steady, fully turbulent flows in approximate local equilibrium. The purpose of this paper is to report on the development of a turbulence model suited for use in arterial flows. The model is of the two-equation eddy-viscosity variety with dependent variables that are zero-valued at a solid wall and vary linearly with distance from it. The effects of transition are introduced by coupling this model to the local value of the intermittency and obtaining the latter from the solution of a modeled transport equation. Comparisons with measurements obtained in oscillatory transitional flows in circular tubes show that the model produces substantial improvements over existing closures. Further pulsatile-flow predictions, driven by a mean-flow wave form obtained in a diseased human carotid artery, indicate that the intermittency-modified model yields much reduced levels of wall shear stress compared to the original, unmodified model. This result, which is attributed to the rapid growth in the thickness of the viscous sublayer arising from the severe acceleration of systole, argues in favor of the use of the model for the prediction of arterial flows.
publisherThe American Society of Mechanical Engineers (ASME)
titleA Turbulence Model for Pulsatile Arterial Flows
typeJournal Paper
journal volume126
journal issue5
journal titleJournal of Biomechanical Engineering
identifier doi10.1115/1.1798032
journal fristpage578
journal lastpage584
identifier eissn1528-8951
keywordsFlow (Dynamics)
keywordsTurbulence
keywordsStress
keywordsReynolds number
keywordsEquations
keywordsViscosity
keywordsEddies (Fluid dynamics)
keywordsShear (Mechanics) AND Measurement
treeJournal of Biomechanical Engineering:;2004:;volume( 126 ):;issue: 005
contenttypeFulltext


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