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    The Asymmetry of Transient Response in Compression Versus Release for Cartilage in Unconfined Compression

    Source: Journal of Biomechanical Engineering:;2001:;volume( 123 ):;issue: 005::page 519
    Author:
    L. P. Li
    ,
    M. D. Buschmann
    ,
    A. Shirazi-Adl
    DOI: 10.1115/1.1388295
    Publisher: The American Society of Mechanical Engineers (ASME)
    Abstract: Observations in compression tests of articular cartilage have revealed unequal load increments for compression and release of the same amplitude applied to a disk with an identical previously imposed compression (in equilibrium). The mechanism of this asymmetric transient response is investigated here using a nonlinear fibril-reinforced model. It is found that the asymmetry is predominantly produced by the fibril stiffening with its tensile strain. In addition, allowing the hydraulic permeability to decrease significantly with compressive dilatation of cartilage increases the transient fibril strain, resulting in a stronger asymmetry. Large deformation also enhances the asymmetry as a consequence of stronger fibril stiffening.
    keyword(s): Stress , Transients (Dynamics) , Compression , Cartilage , Permeability , Disks AND Deformation ,
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      The Asymmetry of Transient Response in Compression Versus Release for Cartilage in Unconfined Compression

    URI
    http://yetl.yabesh.ir/yetl1/handle/yetl/124782
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    • Journal of Biomechanical Engineering

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    contributor authorL. P. Li
    contributor authorM. D. Buschmann
    contributor authorA. Shirazi-Adl
    date accessioned2017-05-09T00:04:11Z
    date available2017-05-09T00:04:11Z
    date copyrightOctober, 2001
    date issued2001
    identifier issn0148-0731
    identifier otherJBENDY-26190#519_1.pdf
    identifier urihttp://yetl.yabesh.ir/yetl/handle/yetl/124782
    description abstractObservations in compression tests of articular cartilage have revealed unequal load increments for compression and release of the same amplitude applied to a disk with an identical previously imposed compression (in equilibrium). The mechanism of this asymmetric transient response is investigated here using a nonlinear fibril-reinforced model. It is found that the asymmetry is predominantly produced by the fibril stiffening with its tensile strain. In addition, allowing the hydraulic permeability to decrease significantly with compressive dilatation of cartilage increases the transient fibril strain, resulting in a stronger asymmetry. Large deformation also enhances the asymmetry as a consequence of stronger fibril stiffening.
    publisherThe American Society of Mechanical Engineers (ASME)
    titleThe Asymmetry of Transient Response in Compression Versus Release for Cartilage in Unconfined Compression
    typeJournal Paper
    journal volume123
    journal issue5
    journal titleJournal of Biomechanical Engineering
    identifier doi10.1115/1.1388295
    journal fristpage519
    journal lastpage522
    identifier eissn1528-8951
    keywordsStress
    keywordsTransients (Dynamics)
    keywordsCompression
    keywordsCartilage
    keywordsPermeability
    keywordsDisks AND Deformation
    treeJournal of Biomechanical Engineering:;2001:;volume( 123 ):;issue: 005
    contenttypeFulltext
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    DSpace software copyright © 2002-2015  DuraSpace
    نرم افزار کتابخانه دیجیتال "دی اسپیس" فارسی شده توسط یابش برای کتابخانه های ایرانی | تماس با یابش
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