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contributor authorC. M. Rodkiewicz
contributor authorS. Zajac
contributor authorJ. Centkowski
date accessioned2017-05-08T23:37:40Z
date available2017-05-08T23:37:40Z
date copyrightNovember, 1992
date issued1992
identifier issn0148-0731
identifier otherJBENDY-25891#527_1.pdf
identifier urihttp://yetl.yabesh.ir/yetl/handle/yetl/109821
description abstractAn elastic model of the arterial system has been used in which a specially designed pumping unit simulated the heart action. Physiological pressures and normal geometry, size, and flow distribution together with the normal cardiac output and use of prosthetic heart valves are the features of the system. Atherosclerosis was simulated by introducing blockages of known cross-section at specific sites of predilection. It has been shown that, for some specific occlusion magnitude in the left or right subclavian, or in the brachycephalic arteries, the stagnant no blood flow condition will appear in the left vertebral, or the right vertebral, or right common carotid, or the right internal carotid arteries. For larger occlusions the blood flow in these arteries reverses its direction, i.e., the “steal syndrome” appears. It is shown that besides the known single steal syndrome there exists also a double steal syndrome, i.e., blood reverses its flow direction simultaneously in two arteries, both on the right side of the arterial system. This blood is taken from the circle of Willis, which at the same time is significantly supplemented by the increased blood flow through the other arteries leading into the circle of Willis.
publisherThe American Society of Mechanical Engineers (ASME)
titleOn the Subclavian Steal Syndrome In Vitro Studies
typeJournal Paper
journal volume114
journal issue4
journal titleJournal of Biomechanical Engineering
identifier doi10.1115/1.2894106
journal fristpage527
journal lastpage532
identifier eissn1528-8951
keywordsFlow (Dynamics)
keywordsBlood
keywordsGeometry
keywordsHeart valve prostheses
keywordsPhysiology
keywordsAtherosclerosis
keywordsCarotid arteries AND Blood flow
treeJournal of Biomechanical Engineering:;1992:;volume( 114 ):;issue: 004
contenttypeFulltext


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